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List of Publications via PubMed (NIH National Library of Medicine) Highlighted Publications Rao, S.D. & Weiss, J.H. Excitotoxic and oxidative cross-talk between motor neurons and glia in ALS pathogenesis. Trends Neurosci. 27 (1) 17-23 (2004). Ogoshi, F. & Weiss, J.H. Heterogeneity of Ca2+-permeable AMPA/kainate channel expression in hippocampal pyramidal neurons: Fluorescence imaging and immunocytochemical assessment. J. Neurosci. 23(33):10521-30 (2003). Sensi, S.L., Ton-That, D., Sullivan, P.G., Jonas, E.A., Gee, K.R., Kaczmarek, L.K. and Weiss, J.H. Modulation of mitochondrial function by endogenous Zn2+ pools. Proc. Natl. Acad. Sci. (USA) 100, 6157-6162 (2003). Rao, S.D., Yin, H.Z. and Weiss, J.H. Disruption of glial glutamate transport by reactive oxygen species produced in motor neurons. J. Neurosci. 23, 2627-2633 (2003). Jia, Y.S., Jeng, J.M., Sensi, S.L. and Weiss, J.H. Zn2+ currents are mediated by Ca2+-permeable AMPA/kainate channels in cultured hippocampal neurons.J. Physiol. 543.1, 35-48 (2002). Sensi, S.L., Ton-That, D. and Weiss J..H. Mitochondrial sequestration and Ca2+ dependent release of cytosolic Zn2+ loads in cortical neurons. Neurobiol. Dis. 10, 100-108 (2002). Yin, H.Z., Sensi, S.L., Ogoshi, F. and Weiss, J.H. Blockade of Ca2+ permeable AMPA/kainate channels decreases oxygen glucose deprivation induced Zn2+ accumulation and neuronal loss in hippocampal pyramidal neurons. J. Neurosci. 22, 1273-9 (2002). Jiang, D., Sullivan, P.G., Sensi, S.L., Steward, O. and Weiss, J.H. Zn2+ induces permeability transition pore opening and release of pro-apoptotic peptides from neuronal mitochondria. J. Biol. Chem. 276, 47524-47529 (2001). Sensi, S.L., Yin, H.Z. and Weiss, J.H. Kainate triggered Zn2+ entry into cortical neurons induces mitochondrial Zn2+ uptake and persistent mitochondrial dysfunction.Eur. J. Neurosci., 2000, 12, 3813-3818. Weiss, J.H. and Sensi, S.S. and Koh, J.Y. Zn2+: Possible potent ionic mediator of neural injury in brain disease. Trends Pharmacol. Sci., 2000, 21, 395-401. Weiss, J.H. and Sensi, S.S. Ca2+ permeable AMPA/kainate channels and selective neurodegeneration.Trends in Neuroscience, 2000, 23, 365-371. Carriedo, S.G., Sensi, S.L., Yin, H.Z. and Weiss, J.H. AMPA exposures induce mitochondrial Ca2+ overload and ROS generation in spinal motor neurons in vitro. Journal of Neuroscience, 2000 20:240-250. Sensi, S.L., Yin, H.Z., Carriedo, S.G., Rao, SS and Weiss, J.H. Preferential Zn2+ influx through Ca2+ permeable AMPA/kainate channels triggers prolonged mitochondrial superoxide production. Proceedings of the National Academy of Science, (USA) 96, 1999; 2414-2419. Yin HZ, Sensi SL, Carriedo SG, Weiss JH Dendritic localization of Ca(2+)-permeable AMPA/kainate channels in hippocampal pyramidal neurons. Journal of Comparative Neurology, 1999 Jun 28;409(2):250-60. Ha, D.H., Robertson, R.T. and Weiss, J.H., Basal forebrain neurons enhance morphological features of a subset of cortical neurons in vitro. Journal of Neuroscience, 18, 4201-4215 (1998). Carriedo, S. G., Yin, H. Z., Sensi, S.L. and Weiss, J. H., Rapid Ca2+ entry through Ca2+-permeable AMPA/kainate channels triggers marked intracellular Ca2+ rises and consequent oxygen radical production. Journal of Neuroscience, 18, 7727-7738 (1998). Carriedo, S.G., Yin, H.Z., and Weiss, J.H. Motor neurons are selectively vulnerable to AMPA/kainate receptor-mediated injury in vitro. Journal of Neuroscience, 16, 4069-4079 (1996). Ha, D.H., Robertson, R.T., Ribak, C.E. and Weiss, J.H., Basal forebrain cholinergic neurons that contact cortical cells in culture display enhanced morphological features and decreased dependence on nerve growth factor. Journal of Comparative Neurology, 373, 451-465 (1996). Lu, Y.M., Yin, H.Z. and Weiss, J.H Ca2+-permeable AMPA/kainate and NMDA channels: High rate of Ca2+ influx underlies potent induction of injury. Journal of Neuroscience, 16, 5457-5465 (1996). Yin, H.Z. and Weiss, J.H. Zn2+ permeates Ca2+ permeable AMPA/kainate channels and triggers selective neural injury. NeuroReport. 6, 2553-2556 (1995). Weiss, J.H., Pike, C.J. and Cotman, C.W. Ca2+ channel blockers attenuate þ-amyloid peptide toxicity to cortical neurons in culture, Journal of Neurochemistry, 62, 372-375 (1994). Yin, H., Turetsky, D., Choi, D.W. and Weiss, J.H. Cortical neurons with Ca2+ permeable AMPA/kainate channels display distinct receptor immunoreactivity and are GABAergic, Neurobiol. Dis,. 1, 43-49 (1994). |
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