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| Dr. Baram's Highlighted Publications (cont.) |
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Highlighted Publications (continued): ...Developmental Neurobiology of stress, and the cellular machinery of CRH Avishai-Eliner S, Brunson KL, Sandman CA, Baram TZ ‘Stressed out?’ Or in (utero). Trends Neurosci, 25:518-24; 2002. Roozendaal B, Brunson KL, Holloway B, McGaugh JL, Baram TZ. Involvement of stress-released Corticotropin-Releasing Hormone in the basolateral amygdala in regulating memory consolidation. Proc Natl Acad Sci, 99:13908-13; 2002. Avishai-Eliner S, Eghbal-Ahmadi M, Tabatchnik E, Brunson KL, Baram TZ. Down-regulation of hypothalamic corticotropin-releasing hormone messenger ribonucleic acid (mRNA) precedes early-life experience-induced changes in hippocampal glucocorticoid receptor mRNA. Endocrinology, 142:89-97; 2001. Avishai-Eliner S, Gilles EE, Eghbal-Ahmadi M, Bar-El Y, Baram TZ. Altered regulation of Gene and Protein Expression of Hypothalamic-Pituitary-Adrenal Axis Components in an immature Rat Model of Chronic Stress. J Neuroendocrinol, 13:799-807; 2001. Brunson KL, Eghbal-Ahmadi M, Roland R, Chen Y, Baram TZ. Long-term progressive hippocampal cell loss and dysfunction induced by early-life administration of corticotropin releasing hormone reproduce the effects of early-life stress. Proc Natl Acad Sci, 98:8856-61; 2001. Brunson KL, Khan N, Eghbal-Ahmadi, Baram TZ. ACTH acts directly on amygdala neurons to down-regulate corticotropin releasing hormone gene expression. Ann Neurol, 49:304-13; 2001. Chen Y, Bender R, Frotscher M, Baram TZ. Novel and transient populations of corticotropin-releasing hormone-expressing neurons in developing hippocampus suggest unique functional roles: a quantitative spatio-temporal analysis. J Neurosci, 21:7171-81; 2001. Eghbal-Ahmadi M, Avishai-Eliner S, Hatalski CG, Baram TZ. Differential regulation of the expression of corticotropin releasing factor receptor type 2 (CRF2) in hypothalamus and amygdala of the immature rat by sensory input and food intake. J Neurosci, 19:3982-91; 1999. Baram TZ, Hatalski CG. Neuropeptide-mediated excitability: A key triggering mechanism for seizure generation in the developing brain. Trends Neurosci 21; 471-76, 1998. |
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